Kainic Acid Impairs the Memory Behavior of APP23 Mice by Increasing Brain Amyloid Load through a Tumor Necrosis Factor-α-Dependent Mechanism-Reference-Cited by-同舟云学术

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Kainic Acid Impairs the Memory Behavior of APP23 Mice by Increasing Brain Amyloid Load through a Tumor Necrosis Factor-α-Dependent Mechanism

Published:2018-06-08 Issue:1 Volume:64 Page:103-116
ISSN:1387-2877
Container-title:Journal of Alzheimer's Disease
language:
Short-container-title:JAD

Author:

Ruan Yang¹,Guo Shi-Jie²,Wang Xu³,Dong Dong⁴,Shen Dong-Hui³,Zhu Jie³⁵,Zheng Xiang-Yu³

Affiliation:

1. Key Laboratory of Pathobiology, Ministry of Education, College of Basic Medical Sciences, Jilin University, Changchun, China

2. Department of Neonatology, The First Hospital of Jilin University, Changchun, China

3. Department of Neurology and Neuroscience Center, The First Hospital of Jilin University, Changchun, China

4. Department of Radiology, The First Hospital of Jilin University, Changchun, China

5. Department of Neurobiology, Care Sciences and Society, Karolinska Institute, Stockholm, Sweden

Publisher

IOS Press

Subject

Psychiatry and Mental health,Geriatrics and Gerontology,Clinical Psychology,General Medicine,General Neuroscience

Reference55 articles.

1. Kainic acid-induced neurodegenerative model: Potentials and limitations;Zheng;J Biomed Biotechnol,2011

2. Selective degeneration and synaptic reorganization of hippocampal interneurons in a chronic model of temporal lobe epilepsy;Ratte;Adv Neurol,2006

3. Use of excitotoxins to lesion the hippocampus: Update;Jarrard;Hippocampus,2002

4. Expression of adenomatous polyposis coli protein in reactive astrocytes in hippocampus of kainic acid-induced rat;Lee;Neurochem Res,2010

5. Differences in two mice strains on kainic acid-induced amygdalar seizures;Kasugai;Biochem Biophys Res Commun,2007

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Kainic acid Induces production and aggregation of amyloid β-protein and memory deficits by activating inflammasomes in NLRP3- and NF-κB-stimulated pathways;Aging;2019-06-10

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