Intracoronary platelet and monocyte activation status within platelet-monocyte complexes are determinants of inflammation in ST elevation myocardial infarction1
Author:
Affiliation:
1. Department of Cardiology, Royal Free Hospital, London, UK
2. Department of Haematology, Royal Free Hospital, London, UK
Publisher
IOS Press
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine,Hematology,Physiology
Reference25 articles.
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3. Monocyte functional responsiveness after PSGL-1-mediated platelet adhesion is dependent on platelet activation status;Bournazos;Arterioscler Thromb Vasc Biol,2008
4. Braune S. , Groß M. , Walter M., Zhou S., Dietze S., Rutschow S., Lendlein A., Tschöpe C. and Jung F., Adhesion and activation of platelets from subjects with coronary artery disease and apparently healthy individuals on biomaterials. J Biomed Mater Res B Appl Biomater (2015), DOI: 10.1002/jbm.b.33366.
5. Neutrophil-platelet adhesion: Relative roles of platelet P-selectin and neutrophil beta2 (DC18) integrins;Brown;Am J Respir Cell Mol Biol,1998
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