SGK1 up-regulates Orai1 expression and VSMC migration during neointima formation after arterial injury-Reference-Cited by-同舟云学术

SGK1 up-regulates Orai1 expression and VSMC migration during neointima formation after arterial injury

Published:2017 Issue:05 Volume:117 Page:1002-1005
ISSN:0340-6245
Container-title:Thrombosis and Haemostasis
language:en
Short-container-title:Thromb Haemost

Author:

Alesutan Ioana,Voelkl Jakob,Geue Sascha,Münzer Patrick,Rodríguez José,Kuhl Dietmar,Lang Florian,Walker-Allgaier Britta,Schaub Malte,Gawaz Meinrad,Borst Oliver

Abstract

Supplementary Material to this article is available online at www.thrombosis-online.com

Publisher

Georg Thieme Verlag KG

Subject

Hematology

Link

http://www.thieme-connect.de/products/ejournals/pdf/10.1160/TH16-09-0690.pdf

Reference23 articles.

1. Interruption of classic CD40L-CD40 signalling but not of the novel CD40L-Mac-1 interaction limits arterial neointima formation in mice

2. Deficiency of MAPK-activated protein kinase 2 (MK2) prevents adverse remodelling and promotes endothelial healing after arterial injury

3. Primate Smooth Muscle Cell Migration From Aortic Explants Is Mediated by Endogenous Platelet-Derived Growth Factor and Basic Fibroblast Growth Factor Acting Through Matrix Metalloproteinases 2 and 9

4. Disruption of Platelet-Derived Growth Factor–Dependent Phosphatidylinositol 3-Kinase and Phospholipase Cγ 1 Activity Abolishes Vascular Smooth Muscle Cell Proliferation and Migration and Attenuates Neointima Formation In Vivo

5. PI3K/p110 inhibition selectively interferes with arterial thrombosis and neointima formation, but not re-endothelialization: potential implications for drug-eluting stent design

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