TAK1 inhibition by natural cyclopeptide RA-V promotes apoptosis and inhibits protective autophagy in Kras-dependent non-small-cell lung carcinoma cells-Reference-Cited by-同舟云学术

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TAK1 inhibition by natural cyclopeptide RA-V promotes apoptosis and inhibits protective autophagy in Kras-dependent non-small-cell lung carcinoma cells

Published:2018 Issue:41 Volume:8 Page:23451-23458
ISSN:2046-2069
Container-title:RSC Advances
language:en
Short-container-title:RSC Adv.

Author:

Yang Jianhong¹²³⁴⁵,Yang Tao¹²³⁴⁵,Yan Wei¹²³⁴⁵,Li Dan¹²³⁴⁵,Wang Fang¹²³⁴⁵,Wang Zhe⁶⁷⁸⁹¹⁰,Guo Yingjie¹¹¹²¹³¹⁰,Bai Peng¹²³⁴⁵,Tan Ninghua⁶⁷⁸⁹¹⁰^ORCID,Chen Lijuan¹²³⁴⁵^ORCID

Affiliation:

1. Cancer Center

2. West China Hospital

3. Sichuan University

4. Collaborative Innovation Center for Biotherapy

5. Chengdu

6. School of Traditional Chinese Pharmacy

7. State Key Laboratory of Natural Medicines

8. China Pharmaceutical University

9. Nanjing

10. China

11. Department of Cardiology

12. The Second Affiliated Hospital of Guangdong Medical University

13. Zhanjiang

Abstract

TAK1 kinase is required for the survival of Kras-dependent non-small-cell lung carcinoma (NSCLC) cells.

Funder

National Natural Science Foundation of China

Chinese Academy of Sciences

China Pharmaceutical University

Ministry of Education of the People's Republic of China

State Administration of Foreign Experts Affairs

Publisher

Royal Society of Chemistry (RSC)

Subject

General Chemical Engineering,General Chemistry

Link

http://pubs.rsc.org/en/content/articlepdf/2018/RA/C8RA04241A

Reference32 articles.

1. Targeting RAS signalling pathways in cancer therapy

2. Implications for KRAS status and EGFR-targeted therapies in metastatic CRC

3. TAK1 Inhibition Promotes Apoptosis in KRAS-Dependent Colon Cancers

4. Autophagy and Metabolism

5. Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis

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