TAK1 Inhibition Promotes Apoptosis in KRAS-Dependent Colon Cancers-Reference-Cited by-同舟云学术

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TAK1 Inhibition Promotes Apoptosis in KRAS-Dependent Colon Cancers

Published:2012-02 Issue:4 Volume:148 Page:639-650
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Singh Anurag,Sweeney Michael F.,Yu Min,Burger Alexa,Greninger Patricia,Benes Cyril,Haber Daniel A.,Settleman Jeff

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference36 articles.

1. Systematic RNA interference reveals that oncogenic KRAS-driven cancers require TBK1;Barbie;Nature,2009

2. Functional interaction of beta-catenin with the transcription factor LEF-1;Behrens;Nature,1996

3. A comparison of normalization methods for high density oligonucleotide array data based on variance and bias;Bolstad;Bioinformatics,2003

4. A versatile viral system for expression and depletion of proteins in mammalian cells;Campeau;PLoS ONE,2009

5. DAVID: Database for Annotation, Visualization, and Integrated Discovery;Dennis;Genome Biol.,2003

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1. TAK1 expression is associated with increased PD-L1 and decreased cancer-specific survival in microsatellite-stable colorectal cancer;Translational Oncology;2024-10

2. Fibroblasts Promote Resistance to KRAS Silencing in Colorectal Cancer Cells;Cancers;2024-07-20

3. Senataxin deficiency disrupts proteostasis through nucleolar ncRNA-driven protein aggregation;Journal of Cell Biology;2024-05-08

4. KRAS silencing alters chromatin physical organization and transcriptional activity in colorectal cancer cells;2024-04-16

5. MEK-mediated CHPF2 phosphorylation promotes colorectal cancer cell proliferation and metastasis by activating NF-κB signaling;Cancer Letters;2024-03

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