Magnolol dimer-derived fragments as PPARγ-selective probes-Reference-Cited by-同舟云学术

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Magnolol dimer-derived fragments as PPARγ-selective probes

Published:2018 Issue:38 Volume:16 Page:7019-7028
ISSN:1477-0520
Container-title:Organic & Biomolecular Chemistry
language:en
Short-container-title:Org. Biomol. Chem.

Author:

Dreier Dominik¹²³⁴^ORCID,Resetar Mirta⁵⁶⁷⁴^ORCID,Temml Veronika⁸⁹¹⁰⁴^ORCID,Rycek Lukas¹²³⁴^ORCID,Kratena Nicolas¹²³⁴^ORCID,Schnürch Michael¹²³⁴^ORCID,Schuster Daniela¹¹⁹¹⁰⁴¹²^ORCID,Dirsch Verena M.⁵⁶⁷⁴^ORCID,Mihovilovic Marko D.¹²³⁴^ORCID

Affiliation:

1. Institute of Applied Synthetic Chemistry

2. TU Wien

3. A-1060 Vienna

4. Austria

5. Department of Pharmacognosy

6. University of Vienna

7. 1090 Vienna

8. Institute of Pharmacy/Pharmacognosy and Center for Molecular Biosciences Innsbruck

9. University of Innsbruck

10. 6020 Innsbruck

11. Institute of Pharmacy/Pharmaceutical Chemistry and Center for Molecular Biosciences Innsbruck

12. Paracelsus Medical University Salzburg

Abstract

Sesqui magnolol A & B have been found to be selective partial PPARγ agonists while truncated magnolol dimer acts as an antagonist.

Funder

Austrian Science Fund

Publisher

Royal Society of Chemistry (RSC)

Subject

Organic Chemistry,Physical and Theoretical Chemistry,Biochemistry

Link

http://pubs.rsc.org/en/content/articlepdf/2018/OB/C8OB01745J

Reference28 articles.

1. The Many Faces of PPARγ

2. PPARγ Is Required for the Differentiation of Adipose Tissue In Vivo and In Vitro

3. Minireview: Challenges and Opportunities in Development of PPAR Agonists

4. PPARs in obesity-induced T2DM, dyslipidaemia and NAFLD

5. Safety of PPAR Agonists

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3. Identification of Plant Phenolics as Novel Pparγ Agonists and Hypoglycemic Agents;2024

4. Total Synthesis of Natural Products: Case Studies in the Evaluation of New Synthetic Methods, Structural Elucidation and Drug Discovery;Chemické listy;2022-04-15

5. Magnolol may contribute to barrier function improvement on imiquimod‑induced psoriasis‑like dermatitis animal model via the downregulation of interleukin‑23;Experimental and Therapeutic Medicine;2021-03-01

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