A metabolomics study of the inhibitory effect of 17-beta-estradiol on osteoclast proliferation and differentiation-Reference-Cited by-同舟云学术

A metabolomics study of the inhibitory effect of 17-beta-estradiol on osteoclast proliferation and differentiation

Published:2015 Issue:2 Volume:11 Page:635-646
ISSN:1742-206X
Container-title:Molecular BioSystems
language:en
Short-container-title:Mol. BioSyst.

Author:

Liu Xiaoyan¹²³⁴⁵,Liu Yanqiu¹²³⁴⁵,Cheng Mengchun¹²³⁴⁵,Zhang Xiaozhe¹²³⁴⁵,Xiao Hongbin¹²³⁴⁵

Affiliation:

1. Key Laboratory of Separation Science for Analytical Chemistry

2. Dalian Institute of Chemical Physics

3. Chinese Academy of Science

4. Dalian 116023

5. China

Abstract

A metabolomics approach was used to explore metabolic alterations during estradiol induced inhibition of osteoclast (RAW 264.7) proliferation and differentiation.

Publisher

Royal Society of Chemistry (RSC)

Subject

Molecular Biology,Biotechnology

Link

http://pubs.rsc.org/en/content/articlepdf/2015/MB/C4MB00528G

Reference68 articles.

1. The 17-β-oestradiol inhibits osteoclast activity by increasing the cannabinoid CB2 receptor expression

2. Estradiol inhibits adhesion and promotes apoptosis in murine osteoclasts in vitro

3. 17?-estradiol downregulates ?3-integrin expression in differentiating and mature human osteoclasts

4. Direct effects of estrogen on differentiation and apoptosis of osteoclasts

5. Estrogen’s bone-protective effects may involve differential IL-1 receptor regulation in human osteoclast-like cells

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