Astaxanthin attenuates d-galactose-induced brain aging in rats by ameliorating oxidative stress, mitochondrial dysfunction, and regulating metabolic markers-Reference-Cited by-同舟云学术

Astaxanthin attenuates d-galactose-induced brain aging in rats by ameliorating oxidative stress, mitochondrial dysfunction, and regulating metabolic markers

Published:2020 Issue:5 Volume:11 Page:4103-4113
ISSN:2042-6496
Container-title:Food & Function
language:en
Short-container-title:Food Funct.

Author:

Liu Han¹²³⁴⁵,Zhang Xuguang⁶⁷⁴⁵,Xiao Jie¹²³⁴⁵^ORCID,Song Mingyue¹²³⁴⁵,Cao Yong¹²³⁴⁵^ORCID,Xiao Hang⁸⁹¹⁰¹¹^ORCID,Liu Xiaojuan¹²³⁴⁵^ORCID

Affiliation:

1. Guangdong Provincial Key Laboratory of Nutraceuticals and Functional Foods

2. College of Food Science

3. South China Agricultural University

4. Guangzhou

5. China

6. Science and Technology Centre

7. By-Health Co. Ltd

8. Department of Food Science

9. University of Massachusetts Amherst

10. Amherst

11. USA

Abstract

Astaxanthin (AX) is a red-colored xanthophyll carotenoid with potent antioxidant, anti-inflammatory, and neuroprotective properties.

Funder

Natural Science Foundation of Guangdong Province

Publisher

Royal Society of Chemistry (RSC)

Subject

General Medicine,Food Science

Link

http://pubs.rsc.org/en/content/articlepdf/2020/FO/D0FO00633E

Reference48 articles.

1. Systemic administration of catalpol prevents d-galactose induced mitochondrial dysfunction in mice

2. Inhibitory Effects of Walnut (Juglans regia) Peptides on Neuroinflammation and Oxidative Stress in Lipopolysaccharide-Induced Cognitive Impairment Mice

3. Oxidation scrutiny in persuaded aging and chronological aging at systemic redox homeostasis level

4. d-Galactose-induced accelerated aging model: an overview

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