Bioengineering of BRAF and COX2 inhibitor nanogels to boost the immunotherapy of melanoma<i>via</i>pyroptosis-Reference-Cited by-同舟云学术

Bioengineering of BRAF and COX2 inhibitor nanogels to boost the immunotherapy of melanomaviapyroptosis

Published:2023 Issue:7 Volume:59 Page:932-935
ISSN:1359-7345
Container-title:Chemical Communications
language:en
Short-container-title:Chem. Commun.

Author:

Zhang Meng-Jie¹,Liang Meng-Yun²,Yang Shao-Chen¹,Ma Xian-Bin²,Wan Shu-Cheng¹,Yang Qi-Chao¹,Wang Shuo¹,Xu Zhigang²^ORCID,Sun Zhi-Jun¹^ORCID

Affiliation:

1. The State Key Laboratory Breeding Base of Basic Science of Stomatology (Hubei-MOST) & Key Laboratory of Oral Biomedicine Ministry of Education, School & Hospital of Stomatology, Wuhan University, Wuhan 430079, P. R. China

2. School of Materials and Energy & Chongqing Engineering Research Center for Micro-Nano Biomedical Materials and Devices, Southwest University, Chongqing 400715, P. R. China

Abstract

CDNPs, combined with BRAFi and Cox2i, induced pyroptosis to enhance cancer immunotherapy and act synergistically with anti-PD-1 therapy.

Funder

National Natural Science Foundation of China

Fundamental Research Funds for the Central Universities

Publisher

Royal Society of Chemistry (RSC)

Subject

Materials Chemistry,Metals and Alloys,Surfaces, Coatings and Films,General Chemistry,Ceramics and Composites,Electronic, Optical and Magnetic Materials,Catalysis

Link

http://pubs.rsc.org/en/content/articlepdf/2023/CC/D2CC05498A

Reference23 articles.

1. Uveal melanoma

2. A decade of checkpoint blockade immunotherapy in melanoma: understanding the molecular basis for immune sensitivity and resistance

3. Antitumor activity of the ERK inhibitor SCH722984 against BRAF mutant, NRAS mutant and wild-type melanoma

4. K27-linked ubiquitination of BRAF by ITCH engages cytokine response to maintain MEK-ERK signaling

5. Targeting Oncogenic Drivers and the Immune System in Melanoma

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