Protection effect of nicotinamide on cardiomyoblast hypoxia/re-oxygenation injury: study of cellular mitochondrial metabolism-Reference-Cited by-同舟云学术

Protection effect of nicotinamide on cardiomyoblast hypoxia/re-oxygenation injury: study of cellular mitochondrial metabolism

Published:2016 Issue:7 Volume:12 Page:2257-2264
ISSN:1742-206X
Container-title:Molecular BioSystems
language:en
Short-container-title:Mol. BioSyst.

Author:

Wang He¹²³⁴⁵,Liang Xiaoping¹²³⁴⁵,Luo Guoan¹²³⁴⁵,Ding Mingyu¹²³⁴⁵,Liang Qionglin¹²³⁴⁵

Affiliation:

1. Beijing Key Lab of Microanalytical Methods & Instrumentation

2. Key lab of Chemical Biology (Ministry of Education)

3. Department of Chemistry

4. Tsinghua University

5. Beijing 100084

Abstract

Nicotinamide exerts a protective effect on cardiomyoblasts against hypoxia/re-oxygenation-induced injury through reduction of reactive oxygen species generation via succinate dehydrogenase inhibition.

Funder

Ministry of Science and Technology

China Postdoctoral Science Foundation

National Natural Science Foundation of China

Publisher

Royal Society of Chemistry (RSC)

Subject

Molecular Biology,Biotechnology

Link

http://pubs.rsc.org/en/content/articlepdf/2016/MB/C6MB00108D

Reference35 articles.

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2. The breathing heart — Mitochondrial respiratory chain dysfunction in cardiac disease

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4. Integrated proteomic and metabolomic analysis reveals the NADH-mediated TCA cycle and energy metabolism disorders based on a new model of chronic progressive heart failure

5. NAD+-dependent SIRT1 deactivation has a key role on ischemia–reperfusion-induced apoptosis

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