Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS-Reference-Cited by-同舟云学术

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS

Published:2014-11-05 Issue:7527 Volume:515 Page:431-435
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Chouchani Edward T.,Pell Victoria R.,Gaude Edoardo,Aksentijević Dunja,Sundier Stephanie Y.,Robb Ellen L.,Logan Angela,Nadtochiy Sergiy M.,Ord Emily N. J.,Smith Anthony C.,Eyassu Filmon,Shirley Rachel,Hu Chou-Hui,Dare Anna J.,James Andrew M.,Rogatti Sebastian,Hartley Richard C.,Eaton Simon,Costa Ana S. H.,Brookes Paul S.,Davidson Sean M.,Duchen Michael R.,Saeb-Parsy Kourosh,Shattock Michael J.,Robinson Alan J.,Work Lorraine M.,Frezza Christian,Krieg Thomas,Murphy Michael P.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature13909.pdf

Reference50 articles.

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2. Yellon, D. M. & Hausenloy, D. J. Myocardial reperfusion injury. N. Engl. J. Med. 357, 1121–1135 (2007)

3. Burwell, L. S., Nadtochiy, S. M. & Brookes, P. S. Cardioprotection by metabolic shut-down and gradual wake-up. J. Mol. Cell. Cardiol. 46, 804–810 (2009)

4. Eltzschig, H. K. & Eckle, T. Ischemia and reperfusion–from mechanism to translation. Nature Med. 17, 1391–1401 (2011)

5. Timmers, L. et al. The innate immune response in reperfused myocardium. Cardiovasc. Res. 94, 276–283 (2012)

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