Identification of Id1 as a downstream effector for arsenic-promoted angiogenesis via PI3K/Akt, NF-κB and NOS signaling-Reference-Cited by-同舟云学术

Identification of Id1 as a downstream effector for arsenic-promoted angiogenesis via PI3K/Akt, NF-κB and NOS signaling

Published:2016 Issue:1 Volume:5 Page:151-159
ISSN:2045-452X
Container-title:Toxicology Research
language:en
Short-container-title:Toxicol. Res.

Author:

Tsai Chun-Hao¹²³⁴⁵,Yang Ming-Hui¹²³⁴⁵,Hung Amos C.¹²³⁴⁵,Wu Shou-Cheng⁶⁷⁸⁵,Chiu Wen-Chin⁹¹⁰²³⁴,Hou Ming-Feng¹¹²³⁴⁵,Tyan Yu-Chang¹²³⁴⁵,Wang Yun-Ming⁶⁷⁸⁵,Yuan Shyng-Shiou F.¹²³⁴⁵

Affiliation:

1. Translational Research Center

2. Kaohsiung Medical University Hospital

3. Kaohsiung Medical University

4. Kaohsiung

5. Taiwan

6. Department of Biological Science and Technology and Institute of Molecular Medicine and Bioengineering

7. National Chiao Tung University

8. Hsinchu

9. Division of Thoracic Surgery

10. Department of Surgery

11. Cancer Center

Abstract

Exposure to arsenic is known to be a risk factor for various types of cancer. In this study, we report that Id1 mediates arsenic-induced angiogenesis through PI3K/Akt, NF-κB, and nitric oxide synthase (NOS) signaling.

Funder

Kaohsiung Medical University

National Health Research Institutes

Ministry of Health and Welfare

Publisher

Oxford University Press (OUP)

Subject

Health, Toxicology and Mutagenesis,Toxicology

Link

http://pubs.rsc.org/en/content/articlepdf/2016/TX/C5TX00280J

Reference69 articles.

1. The Ecology of Arsenic