B cell–specific and stimulation-responsive enhancers derepress Aicda by overcoming the effects of silencers-Reference-Cited by-同舟云学术

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B cell–specific and stimulation-responsive enhancers derepress Aicda by overcoming the effects of silencers

Published:2009-12-06 Issue:2 Volume:11 Page:148-154
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Tran Thinh Huy,Nakata Mikiyo,Suzuki Keiichiro,Begum Nasim A,Shinkura Reiko,Fagarasan Sidonia,Honjo Tasuku,Nagaoka Hitoshi

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/ni.1829.pdf

Reference48 articles.

1. Muramatsu, M. et al. Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme. Cell 102, 553–563 (2000).

2. Revy, P. et al. Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2). Cell 102, 565–575 (2000).

3. Honjo, T., Kinoshita, K. & Muramatsu, M. Molecular mechanism of class switch recombination: linkage with somatic hypermutation. Annu. Rev. Immunol. 20, 165–196 (2002).

4. Stavnezer, J., Guikema, J.E. & Schrader, C.E. Mechanism and regulation of class switch recombination. Annu. Rev. Immunol. 26, 261–292 (2008).

5. Petersen, S. et al. AID is required to initiate Nbs1/γ-H2AX focus formation and mutations at sites of class switching. Nature 414, 660–665 (2001).

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