AID is required to initiate Nbs1/γ-H2AX focus formation and mutations at sites of class switching-Reference-Cited by-同舟云学术

AID is required to initiate Nbs1/γ-H2AX focus formation and mutations at sites of class switching

Published:2001-12-06 Issue:6864 Volume:414 Page:660-665
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Petersen Simone,Casellas Rafael,Reina-San-Martin Bernardo,Chen Hua Tang,Difilippantonio Michael J.,Wilson Patrick C.,Hanitsch Leif,Celeste Arkady,Muramatsu Masamichi,Pilch Duane R.,Redon Christophe,Ried Thomas,Bonner William M.,Honjo Tasuku,Nussenzweig Michel C.,Nussenzweig André

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

https://www.nature.com/articles/414660a.pdf

Reference30 articles.

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2. Carney, J. P. et al. The hMre11/hRad50 protein complex and Nijmegen breakage syndrome: linkage of double-strand break repair to the cellular DNA damage response. Cell 93, 477–486 (1998).

3. Rogakou, E. P., Boon, C., Redon, C. & Bonner, W. M. Megabase chromatin domains involved in DNA double-strand breaks in vivo. J. Cell. Biol. 146, 905–916 (1999).

4. Downs, J. A., Lowndes, N. F. & Jackson, S. P. A role for Saccharomyces cerevisiae histone H2A in DNA repair. Nature 408, 1001–1004 (2000).

5. Revy, P. et al. Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2). Cell 102, 565–575 (2000).

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