The ubiquitin E3 ligase TRAF6 exacerbates pathological cardiac hypertrophy via TAK1-dependent signalling-Reference-Cited by-同舟云学术

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The ubiquitin E3 ligase TRAF6 exacerbates pathological cardiac hypertrophy via TAK1-dependent signalling

Published:2016-06-01 Issue:1 Volume:7 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Ji Yan-Xiao,Zhang Peng,Zhang Xiao-Jing,Zhao Yi-Chao,Deng Ke-Qiong,Jiang Xi,Wang Pi-Xiao,Huang Zan,Li Hongliang

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms11267.pdf

Reference70 articles.

1. Hill, J. A. & Olson, E. N. Cardiac plasticity. N. Engl. J. Med. 358, 1370–1380 (2008).

2. Jiang, D. S. et al. IRF8 suppresses pathological cardiac remodelling by inhibiting calcineurin signalling. Nat. Commun. 5, 3303 (2014).

3. Putinski, C. et al. Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy. Proc. Natl Acad. Sci. USA 110, E4079–E4087 (2013).

4. Xie, P. TRAF molecules in cell signaling and in human diseases. J. Mol. Signal. 8, 7 (2013).

5. Xu, L. G., Li, L. Y. & Shu, H. B. TRAF7 potentiates MEKK3-induced AP1 and CHOP activation and induces apoptosis. J. Biol. Chem. 279, 17278–17282 (2004).

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