The Absence of uPAR Is Associated with the Progression of Dermal Fibrosis-Reference-Cited by-同舟云学术

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The Absence of uPAR Is Associated with the Progression of Dermal Fibrosis

Published:2008-12 Issue:12 Volume:128 Page:2792-2797
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Kanno Yosuke,Kaneiwa Aki,Minamida Misato,Kanno Miho,Tomogane Kanji,Takeuchi Koji,Okada Kiyotaka,Ueshima Shigeru,Matsuo Osamu,Matsuno Hiroyuki

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference27 articles.

1. Reduced collagenase gene expression in fibroblasts from hypertrophic scar tissue;Arakawa;Br J Dermatol,1996

2. Urokinase receptor cleavage: a crucial step in fibroblast-to-myofibroblast differentiation;Bernstein;Mol Biol Cell,2007

3. uPAR: a versatile signalling orchestrator;Blasi;Nat Rev Mol Cell Biol,2002

4. Matrix metalloproteinase 12-dependent cleavage of urokinase receptor in systemic sclerosis microvascular endothelial cells results in impaired angiogenesis;D’Alessio;Arthritis Rheum,2004

5. Generation and characterization of urokinase receptor-deficient mice;Dewerchin;J Clin Invest,1996

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2. The uPA/uPAR System Orchestrates the Inflammatory Response, Vascular Homeostasis, and Immune System in Fibrosis Progression;International Journal of Molecular Sciences;2023-01-16

3. α2-Antiplasmin as a Potential Therapeutic Target for Systemic Sclerosis;Life;2022-03-09

4. Comment on: Iontophoresis of treprostinil promotes wound healing in a murine model of scleroderma-related ulcers;Rheumatology;2022-01-07

5. E4 engages uPAR and enolase-1 and activates urokinase to exert antifibrotic effects;JCI Insight;2021-12-22

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