Helicobacter pylori exploits human CEACAMs via HopQ for adherence and translocation of CagA-Reference-Cited by-同舟云学术

Helicobacter pylori exploits human CEACAMs via HopQ for adherence and translocation of CagA

Published:2016-10-17 Issue:1 Volume:2 Page:
ISSN:2058-5276
Container-title:Nature Microbiology
language:en
Short-container-title:Nat Microbiol

Author:

Königer Verena,Holsten Lea,Harrison Ute,Busch Benjamin,Loell Eva,Zhao Qing,Bonsor Daniel A.,Roth Alexandra,Kengmo-Tchoupa Arnaud,Smith Stella I.,Mueller Susanna,Sundberg Eric J.,Zimmermann Wolfgang^ORCID,Fischer Wolfgang,Hauck Christof R.,Haas Rainer^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Microbiology (medical),Genetics,Applied Microbiology and Biotechnology,Immunology,Microbiology

Link

http://www.nature.com/articles/nmicrobiol2016188.pdf

Reference40 articles.

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2. Peek, R. M. & Blaser, M. J. Helicobacter pylori and gastrointestinal tract adenocarcinomas. Nat. Rev. Cancer 2, 28–37 (2002).

3. Kwok, T. et al. Helicobacter exploits integrin for type IV secretion and kinase activation. Nature 449, 862–866 (2007).

4. Jimenez-Soto, L. F. et al. Helicobacter pylori type IV secretion apparatus exploits beta1 integrin in a novel RGD-independent manner. PLoS Pathog. 5, e1000684 (2009).

5. Odenbreit, S. et al. Translocation of Helicobacter pylori CagA into gastric epithelial cells by type IV secretion. Science 287, 1497–1500 (2000).

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