CAMTA1 is a novel tumour suppressor regulated by miR-9/9*in glioblastoma stem cells

Author:

Schraivogel Daniel1,Weinmann Lasse2,Beier Dagmar34,Tabatabai Ghazaleh5,Eichner Alexander6,Zhu Jia Yun2,Anton Martina7,Sixt Michael6,Weller Michael5,Beier Christoph P34,Meister Gunter12

Affiliation:

1. Biochemistry Center Regensburg (BZR), University of Regensburg; Regensburg Germany

2. Laboratory of RNA Biology, Max-Planck-Institute of Biochemistry; Martinsried Germany

3. Department of Neurology, RWTH Aachen; Aachen Germany

4. Department of Neurology, University of Regensburg; Regensburg Germany

5. Department of Neurology, University Hospital Zurich; Zurich Switzerland

6. IST Austria (Institute of Science and Technology Austria; Klosterneuburg Austria

7. TU Munich, Institute of Experimental Oncology and Therapy Research; Munich Germany

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference68 articles.

1. Allelic losses at 1p36 and 19q13 in gliomas: correlation with histologic classification, definition of a 150-kb minimal deleted region on 1p36, and evaluation of CAMTA1 as a candidate tumor suppressor gene;Barbashina;Clin Cancer Res,2005

2. MicroRNAs: target recognition and regulatory functions;Bartel;Cell,2009

3. CD133(+) and CD133(−) glioblastoma-derived cancer stem cells show differential growth characteristics and molecular profiles;Beier;Cancer Res,2007

4. CD133 expression and cancer stem cells predict prognosis in high-grade oligodendroglial tumors;Beier;Brain Pathol,2008

5. Identification of human microRNA targets from isolated argonaute protein complexes;Beitzinger;RNA Biol,2007

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