Tyrosine dephosphorylation is required for Bak activation in apoptosis-Reference-Cited by-同舟云学术

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Tyrosine dephosphorylation is required for Bak activation in apoptosis

Published:2010-10-19 Issue:22 Volume:29 Page:3853-3868
ISSN:0261-4189
Container-title:The EMBO Journal
language:
Short-container-title:EMBO J

Author:

Fox Joanna L,Ismail Ferina,Azad Abul,Ternette Nicola,Leverrier Sabrina,Edelmann Mariola J,Kessler Benedikt M,Leigh Irene M,Jackson Sarah,Storey Alan

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference64 articles.

1. The Bcl-2 apoptotic switch in cancer development and therapy

2. HPV8 early genes modulate differentiation and cell cycle of primary human adult keratinocytes

3. Bax Is Present as a High Molecular Weight Oligomer/Complex in the Mitochondrial Membrane of Apoptotic Cells

4. Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death

5. MEK and EGFR inhibition demonstrate synergistic activity in EGFR-dependent NSCLC

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1. Retraction Note: MEK inhibition enhances ABT-737-induced leukemia cell apoptosis via prevention of ERK-activated MCL-1 induction and modulation of MCL-1/BIM complex;Leukemia;2024-07-18

2. Kinase signalling adaptation supports dysfunctional mitochondria in disease;Frontiers in Molecular Biosciences;2024-01-26

3. Mathematical modelling of Alzheimer’s disease biomarkers: Targeting Amyloid beta, Tau protein, Apolipoprotein E and Apoptotic pathways;American Journal of Translational Research;2024

4. Phosphoproteomic analysis of duck egg yolk provides novel insights into its characteristics and biofunctions;Journal of the Science of Food and Agriculture;2021-08-18

5. Mitochondrial Kinases and the Role of Mitochondrial Protein Phosphorylation in Health and Disease;Life;2021-01-23

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