The decline in synaptic GluN2B and rise in inhibitory neurotransmission determine the end of a critical period-Reference-Cited by-同舟云学术

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The decline in synaptic GluN2B and rise in inhibitory neurotransmission determine the end of a critical period

Published:2016-09-28 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Isoo Noriko,Ohno Takae,Isowaki Mutsumi,Fukuda Satoshi,Murabe Naoyuki,Mizukami Hiroaki,Ozawa Keiya,Mishina Masayoshi,Sakurai Masaki

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep34196.pdf

Reference44 articles.

1. Hubel, D. H. & Wiesel, T. N. The period of susceptibility to the physiological effects of unilateral eye closure in kittens. J Physiol 206, 419–436 (1970).

2. Hensch, T. K. et al. Local GABA circuit control of experience-dependent plasticity in developing visual cortex. Science 282, 1504–1509 (1998).

3. Colonnese, M. T. & Constantine-Paton, M. Developmental Period for N-Methyl-D-Aspartate (NMDA) Receptor-Dependent Synapse Elimination Correlated with Visuotopic Map Refinement. J Comp Neurol 494, 738–751 (2006).

4. Phillips, M. A. & Constantine-Paton, M. NMDA receptors and development. Developmental Neurobiology, Lemke, G., ed. (La Jolla, CA, USA: Salk Institute for Biological Studies), pp. 438–448 (2009).

5. Fox, K. A critical period for experience-dependent synaptic plasticity in rat barrel cortex. J Neurosci 12, 1826–1838 (1992).

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