Lung-injury depresses glutamatergic synaptic transmission in the nucleus tractus solitarii via discrete age-dependent mechanisms in neonatal rats-Reference-Cited by-同舟云学术

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Lung-injury depresses glutamatergic synaptic transmission in the nucleus tractus solitarii via discrete age-dependent mechanisms in neonatal rats

Published:2018-05 Issue: Volume:70 Page:398-422
ISSN:0889-1591
Container-title:Brain, Behavior, and Immunity
language:en
Short-container-title:Brain, Behavior, and Immunity

Author:

Litvin David G.,Dick Thomas E.,Smith Corey B.,Jacono Frank J.

Funder

School of Medicine

Core

Case Western Reserve University

National Institutes of Health

VA Office of Research and Development

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference177 articles.

1. Enhanced firing in NTS induced by short-term sustained hypoxia is modulated by glia-neuron interaction;Accorsi-Mendonca;J. Neurosci.,2015

2. Conservation of glutamate receptor 2-containing AMPA receptors during long-term potentiation;Adesnik;J. Neurosci.,2007

3. Structural changes in AMPA-receptive neurons in the nucleus of the solitary tract of spontaneously hypertensive rats;Aicher;Hypertension,2003

4. An evaluation of causes for unreliability of synaptic transmission;Allen;Proc. Natl. Acad. Sci. U.S.A.,1994

5. Chronic intermittent hypoxia depresses afferent neurotransmission in NTS neurons by a reduction in the number of active synapses;Almado;J. Neurosci.,2012

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