Author:
Welsh Michael J.,Ostedgaard Lynda S.
Publisher
Springer Science and Business Media LLC
Subject
Genetics,Biochemistry,Structural Biology
Reference20 articles.
1. Molecular mechanisms of CFTR chloride channel dysfunction in cystic fibrosis
2. Solution structure of a protein inhibitor of neuronal nitric oxide synthase
3. Welsh, M.J., Tsui, L.-C., Boat, T.F. & Beaudet, A.L. In The metabolic and molecular basis of inherited disease (eds Scriver, C.R., Beaudet, A.L., Sly, W.S. & Valle, D.) 3799–3876 (McGraw-Hill, Inc., New York; 1995).
4. Defective intracellular transport and processing of CFTR is the molecular basis of most cystic fibrosis
5. Conformational maturation of CFTR but not its mutant counterpart (delta F508) occurs in the endoplasmic reticulum and requires ATP.
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