Partial rescue of Brca15–6 early embryonic lethality by p53 or p21 null mutation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics
Link
http://www.nature.com/articles/ng0797-298.pdf
Reference26 articles.
1. Gowen, L.C., Johnson, B.L., Latour, A.M., Sulik, K.K. & Koller, B.H. Brca1 deficiency results in early embryonic lethality characterized by neuroepithelial abnormalities. Nature Genet. 12, 191–194 (1996).
2. Hakem, R. et al. The tumor suppressor gene Brcal is required for embryonic cellular proliferation in the mouse. Cell 85, 1009–1023 (1996).
3. Liu, C.Y., Fleskennikitin, A., Li, S., Zeng, Y.Y. & Lee, W.H. Inactivation of the mouse Brcal gene leads to failure in the morphogenesis of the egg cylinder in early postimplantation development. Genes Dev. 10, 1835–1843 (1996).
4. Momand, J., Zambetti, G.P., Olson, D.C., George, D. & Levine, A.J. The mdm-2 oncogene product forms a complex with the p53 protein and inhibits p53 mediated transaction. Cell 69, 1237–1245 (1992).
5. Gu, Y., Turek, C.W. & Morgan, D.O. Inhibition of CDK2 activity in vivo by an associated 20k regulatory subunit. Nature 366, 707–710 (1993).
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