S-nitrosylation of c-Jun N-terminal kinase mediates pressure overload-induced cardiac dysfunction and fibrosis-Reference-Cited by-同舟云学术

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S-nitrosylation of c-Jun N-terminal kinase mediates pressure overload-induced cardiac dysfunction and fibrosis

Published:2021-05-19 Issue: Volume: Page:
ISSN:1671-4083
Container-title:Acta Pharmacologica Sinica
language:en
Short-container-title:Acta Pharmacol Sin

Author:

Zhou Miao,Chen Ji-yu,Chao Meng-Lin,Zhang Chao,Shi Zhi-guang,Zhou Xue-chun,Xie Li-ping,Sun Shi-xiu,Huang Zheng-rong,Luo Shan-shan,Ji Yong

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Pharmacology,General Medicine

Link

http://www.nature.com/articles/s41401-021-00674-9.pdf

Reference46 articles.

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2. Lopez B, Gonzalez A, Ravassa S, Beaumont J, Moreno MU, San Jose G, et al. Circulating biomarkers of myocardial fibrosis: the need for a reappraisal. J Am Coll Cardiol. 2015;65:2449–56.

3. Cokkinos DV, Pantos C. Myocardial remodeling, an overview. Heart Fail Rev. 2011;16:1–4.

4. Meng G, Xiao Y, Ma Y, Tang X, Xie L, Liu J, et al. Hydrogen sulfide regulates kruppel-like factor 5 transcription activity via specificity protein 1 S-sulfhydration at Cys664 to prevent myocardial hypertrophy. J Am Heart Assoc. 2016;5:1–17.

5. Meng G, Liu J, Liu S, Song Q, Liu L, Xie L, et al. Hydrogen sulfide pretreatment improves mitochondrial function in myocardial hypertrophy via a SIRT3-dependent manner. Br J Pharmacol. 2018;175:1126–45.

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2. S-nitrosylation of EMMPRIN influences the migration of HSCs and MMP activity in liver fibrosis;Acta Biochimica et Biophysica Sinica;2023-09-01

3. Protein posttranslational modifications in health and diseases: Functions, regulatory mechanisms, and therapeutic implications;MedComm;2023-05-02

4. Construction of atrial fibrillation‐related circRNA/lncRNA‐miRNA‐mRNA regulatory network and analysis of potential biomarkers;Journal of Clinical Laboratory Analysis;2023-01

5. Cardiomyocyte-specific regression of nitrosative stress-mediated S-Nitrosylation of IKKγ alleviates pathological cardiac hypertrophy;Cellular Signalling;2022-10

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