Glycogen shortage during fasting triggers liver–brain–adipose neurocircuitry to facilitate fat utilization-Reference-Cited by-同舟云学术

Glycogen shortage during fasting triggers liver–brain–adipose neurocircuitry to facilitate fat utilization

Published:2013-08-13 Issue:1 Volume:4 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Izumida Yoshihiko,Yahagi Naoya,Takeuchi Yoshinori,Nishi Makiko,Shikama Akito,Takarada Ayako,Masuda Yukari,Kubota Midori,Matsuzaka Takashi,Nakagawa Yoshimi,Iizuka Yoko,Itaka Keiji,Kataoka Kazunori,Shioda Seiji,Niijima Akira,Yamada Tetsuya,Katagiri Hideki,Nagai Ryozo,Yamada Nobuhiro,Kadowaki Takashi,Shimano Hitoshi

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms3316.pdf

Reference16 articles.

1. Reitman, M. L. FGF21: a missing link in the biology of fasting. Cell Metab. 5, 405–407 (2007).

2. Bernal-Mizrachi, C. et al. An afferent vagal nerve pathway links hepatic pparalpha activation to glucocorticoid-induced insulin resistance and hypertension. Cell Metab. 5, 91–102 (2007).

3. Wan, Z. et al. Epinephrine-mediated regulation of PDK4 mRNA in rat adipose tissue. Am. J. Physiol. Cell Ph. 299, C1162–C1170 (2010).

4. Haque, M. S. et al. Role of the sympathetic nervous system and insulin in enhancing glucose uptake in peripheral tissues after intrahypothalamic injection of leptin in rats. Diabetes 48, 1706–1712 (1999).

5. Nakagawa, Y. et al. TFE3 transcriptionally activates hepatic IRS-2, participates in insulin signaling and ameliorates diabetes. Nat. Med. 12, 107–113 (2006).

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