TFE3 transcriptionally activates hepatic IRS-2, participates in insulin signaling and ameliorates diabetes-Reference-Cited by-同舟云学术

TFE3 transcriptionally activates hepatic IRS-2, participates in insulin signaling and ameliorates diabetes

Published:2005-12-04 Issue:1 Volume:12 Page:107-113
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Nakagawa Yoshimi,Shimano Hitoshi,Yoshikawa Tomohiro,Ide Tomohiro,Tamura Mariko,Furusawa Mika,Yamamoto Takashi,Inoue Noriyuki,Matsuzaka Takashi,Takahashi Akimitsu,Hasty Alyssa H,Suzuki Hiroaki,Sone Hirohito,Toyoshima Hideo,Yahagi Naoya,Yamada Nobuhiro

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm1334.pdf

Reference36 articles.

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2. Virkamaki, A., Ueki, K. & Kahn, C.R. Protein-protein interaction in insulin signaling and the molecular mechanisms of insulin resistance. J. Clin. Invest. 103, 931–943 (1999).

3. Withers, D.J. et al. Disruption of IRS-2 causes type 2 diabetes in mice. Nature 391, 900–904 (1998).

4. Kubota, N. et al. Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes 49, 1880–1889 (2000).

5. Rother, K.I. et al. Evidence that IRS-2 phosphorylation is required for insulin action in hepatocytes. J. Biol. Chem. 273, 17491–17497 (1998).

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