FBXL4 mutations cause excessive mitophagy via BNIP3/BNIP3L accumulation leading to mitochondrial DNA depletion syndrome-Reference-Cited by-同舟云学术

FBXL4 mutations cause excessive mitophagy via BNIP3/BNIP3L accumulation leading to mitochondrial DNA depletion syndrome

Published:2023-08-11 Issue:10 Volume:30 Page:2351-2363
ISSN:1350-9047
Container-title:Cell Death & Differentiation
language:en
Short-container-title:Cell Death Differ

Author:

Chen Yingji,Jiao Dongyue,Liu Yang,Xu Xiayun,Wang Yilin,Luo Xiaona,Saiyin Hexige^ORCID,Li Yao^ORCID,Gao Kun^ORCID,Chen Yucai,Zhao Shi-Min^ORCID,Ma Lixiang,Wang Chenji^ORCID

Funder

National Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

https://www.nature.com/articles/s41418-023-01205-1.pdf

Reference29 articles.

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3. Palikaras K, Lionaki E, Tavernarakis N. Mechanisms of mitophagy in cellular homeostasis, physiology and pathology. Nat Cell Biol. 2018;20:1013–22.

4. Misgeld T, Schwarz TL. Mitostasis in Neurons: Maintaining Mitochondria in an Extended Cellular Architecture. Neuron. 2017;96:651–66.

5. Newman LE, Shadel GS. Pink1/Parkin link inflammation, mitochondrial stress, and neurodegeneration. J Cell Biol. 2018;217:3327–9.

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3. PPTC7 antagonizes mitophagy by promoting BNIP3 and NIX degradation via SCFFBXL4;EMBO Reports;2024-07-11

4. Control of mitophagy initiation and progression by the TBK1 adaptors NAP1 and SINTBAD;Nature Structural & Molecular Biology;2024-06-25

5. PPTC7 acts as an essential co-factor of the SCF^FBXL4ubiquitin ligase complex to restrict BNIP3/BNIP3L-dependent mitophagy;2024-06-21