Osteopontin isoforms differentially promote arteriogenesis in response to ischemia via macrophage accumulation and survival-Reference-Cited by-同舟云学术

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Osteopontin isoforms differentially promote arteriogenesis in response to ischemia via macrophage accumulation and survival

Published:2018-06-29 Issue:3 Volume:99 Page:331-345
ISSN:0023-6837
Container-title:Laboratory Investigation
language:en
Short-container-title:Lab Invest

Author:

Lee Grace Sanghee,Salazar Hector F.,Joseph Giji,Lok Zoe Shin Yee,Caroti Courtney M.,Weiss Daiana,Taylor W. Robert,Lyle Alicia N.

Funder

U.S. Department of Health & Human Services | NIH | National Heart, Lung, and Blood Institute

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Pathology and Forensic Medicine

Link

http://www.nature.com/articles/s41374-018-0094-8.pdf

Reference77 articles.

1. Fowkes FG, Aboyans V, Fowkes FJ, et al. Peripheral artery disease: epidemiology and global perspectives. Nat Rev Cardiol. 2016;14:156–70.

2. Scholz D, Ziegelhoeffer T, Helisch A, et al. Contribution of arteriogenesis and angiogenesis to postocclusive hindlimb perfusion in mice. J Mol Cell Cardiol. 2002;34:775–87.

3. Helisch A, Schaper W. Arteriogenesis: the development and growth of collateral arteries. Microcirculation. 2003;10:83–97.

4. Isner JM, Takayuki A. Therapeutic angiogenesis. Front Biosci. 1998;3:e49–69.

5. Sabia PJ, Powers ER, Jayaweera AR, et al. Functional significance of collateral blood flow in patients with recent acute myocardial infarction. A study using myocardial contrast echocardiography. Circulation. 1992;85:2080–9.

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