IVIG pluripotency and the concept of Fc-sialylation: challenges to the scientist-Reference-Cited by-同舟云学术

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IVIG pluripotency and the concept of Fc-sialylation: challenges to the scientist

Published:2014-04-25 Issue:5 Volume:14 Page:349-349
ISSN:1474-1733
Container-title:Nature Reviews Immunology
language:en
Short-container-title:Nat Rev Immunol

Author:

von Gunten Stephan,Shoenfeld Yehuda,Blank Miri,Branch Donald R.,Vassilev Tchavdar,Käsermann Fabian,Bayry Jagadeesh,Kaveri Srini,Simon Hans-Uwe

Publisher

Springer Science and Business Media LLC

Subject

Energy Engineering and Power Technology,Fuel Technology

Link

http://www.nature.com/articles/nri3401-c1.pdf

Reference15 articles.

1. Schwab, I. & Nimmerjahn, F. Intravenous immunoglobulin therapy: how does IgG modulate the immune system? Nature Rev. Immunol. 13, 176–189 (2013).

2. Abe, J. et al. Gene expression profiling of the effect of high-dose intravenous Ig in patients with Kawasaki disease. J. Immunol. 174, 5837–5845 (2005).

3. Maddur, M. S. et al. Inhibition of differentiation, amplification, and function of human TH17 cells by intravenous immunoglobulin. J. Allergy Clin. Immunol. 127, 823–830 (2011).

4. Trinath, J. et al. Intravenous immunoglobulin expands regulatory T cells via induction of cyclooxygenase-2-dependent prostaglandin E2 in human dendritic cells. Blood 122, 1419–1427 (2013).

5. Käsermann, F. et al. Analysis and functional consequences of increased Fab-sialylation of intravenous immunoglobulin (IVIG) after lectin fractionation. PLoS ONE 7, e37243 (2012).

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