Inhibitors of Phosphodiesterase Isoforms III or IV Suppress Islet-Cell Nitric Oxide Production
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Molecular Biology,Pathology and Forensic Medicine
Link
http://www.nature.com/articles/3780323.pdf
Reference39 articles.
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2. Badger AM, Olivera DL, and Esser KM (1994). Beneficial effects of phosphodiesterase inhibitors BRL 61063, pentoxifylline, rolipram in murine models of endotoxic shock. Circ Shock 44: 188–195.
3. Belin VD, Mabley JG, James RF, Swift SM, Clayton HA, Titheradge MA, and Green IC (1999). Glucagon decreases cytokine induction of nitric oxide synthase and action on insulin secretion in RIN5F cells and rat and human islets of Langerhans. Cytokine 11: 585–592.
4. Bergmann L, Kroncke KD, Suschek C, and Kolb-Bachofen V (1992). Cytotoxic action of IL-1β against pancreatic islets is mediated via nitric oxide formation and is inhibited by NG-monomethyl-L-arginine. FEBS Lett 299: 103–106.
5. Beshay E, Croze F, and Prud'homme GJ (2001). The phosphodiesterase inhibitors pentoxifylline and rolipram suppress macrophage activation and nitric oxide production in vitro and in vivo. Clin Immunol 98: 272–279.
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