Amyloid and Nonfibrillar Deposits in Mice Transgenic for Wild-Type Human Transthyretin: A Possible Model for Senile Systemic Amyloidosis-Reference-Cited by-同舟云学术

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Amyloid and Nonfibrillar Deposits in Mice Transgenic for Wild-Type Human Transthyretin: A Possible Model for Senile Systemic Amyloidosis

Published:2001-03-01 Issue:3 Volume:81 Page:385-396
ISSN:0023-6837
Container-title:Laboratory Investigation
language:en
Short-container-title:Lab Invest

Author:

Teng Mei-hui,Yin Jian-yun,Vidal Ruben,Ghiso Jorge,Kumar Asok,Rabenou Rahmin,Shah Alan,Jacobson Daniel R,Tagoe Clement,Gallo Gloria,Buxbaum Joel

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Pathology and Forensic Medicine

Link

http://www.nature.com/articles/3780246.pdf

Reference43 articles.

1. Bales KR, Verina T, Dodel RC, Du Y, Altstiel L, Bender M, Hyslop P, Johnstone EM, Little SP, Cummins DJ, Piccardo P, Ghetti B, and Paul SM (1997). Lack of apolipoprotein E dramatically reduces amyloid beta-peptide deposition. Nat Genet 17: 263–264.

2. Benson MD and Uemichi T (1996). Transthyretin amyloidosis. Amyloid Int J Exp Clin Invest 3: 44–56.

3. Bugiani O, Giaccone G, Frangione B, Ghetti B, and Tagliavini F (1989). Alzheimer patients: Preamyloid deposits are more widely distributed than senile plaques throughout the central nervous system. Neurosci Lett 103: 263–268.

4. Buxbaum JN, Chuba JV, Hellman GC, Solomon A, and Gallo GR (1990). Monoclonal immunoglobulin deposition disease: Light chain and light and heavy chain deposition diseases and their relation to light chain amyloidosis. Clinical features, immunopathology, and molecular analysis. Ann Intern Med 112: 455–464.

5. Buxbaum JN and Tagoe CE (2000). The genetics of the amyloidoses. Annu Rev Med 51: 543–569.

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