The helicase Ded1p controls use of near-cognate translation initiation codons in 5′ UTRs-Reference-Cited by-同舟云学术

The helicase Ded1p controls use of near-cognate translation initiation codons in 5′ UTRs

Published:2018-06-27 Issue:7712 Volume:559 Page:130-134
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Guenther Ulf-Peter,Weinberg David E.,Zubradt Meghan M.,Tedeschi Frank A.,Stawicki Brittany N.,Zagore Leah L.,Brar Gloria A.,Licatalosi Donny D.,Bartel David P.,Weissman Jonathan S.,Jankowsky Eckhard

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41586-018-0258-0.pdf

Reference50 articles.

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2. Bol, G. M., Xie, M. & Raman, V. DDX3, a potential target for cancer treatment. Mol. Cancer 14, 188 (2015).

3. Oh, S. et al. Medulloblastoma-associated DDX3 variant selectively alters the translational response to stress. Oncotarget 7, 28169–28182 (2016).

4. Pugh, T. J. et al. Medulloblastoma exome sequencing uncovers subtype-specific somatic mutations. Nature 488, 106–110 (2012).

5. Snijders Blok, L. et al. Mutations in DDX3X are a common cause of unexplained intellectual disability with gender-specific effects on Wnt signaling. Am. J. Hum. Genet. 97, 343–352 (2015).

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