A myeloid–stromal niche and gp130 rescue in NOD2-driven Crohn’s disease-Reference-Cited by-同舟云学术

A myeloid–stromal niche and gp130 rescue in NOD2-driven Crohn’s disease

Published:2021-03-31 Issue:7858 Volume:593 Page:275-281
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Nayar Shikha^ORCID,Morrison Joshua K.^ORCID,Giri Mamta,Gettler Kyle,Chuang Ling-shiang^ORCID,Walker Laura A.,Ko Huaibin M.^ORCID,Kenigsberg Ephraim^ORCID,Kugathasan Subra^ORCID,Merad Miriam^ORCID,Chu Jaime,Cho Judy H.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41586-021-03484-5.pdf

Reference48 articles.

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2. West, N. R. et al. Oncostatin M drives intestinal inflammation and predicts response to tumor necrosis factor-neutralizing therapy in patients with inflammatory bowel disease. Nat. Med. 23, 579–589 (2017).

3. Martin, J. C. et al. Single-cell analysis of Crohn’s disease lesions identifies a pathogenic cellular module associated with resistance to anti-TNF therapy. Cell 178, 1493–1508.e20 (2019).

4. Ogura, Y. et al. A frameshift mutation in NOD2 associated with susceptibility to Crohn’s disease. Nature 411, 603–606 (2001).

5. Hugot, J. P. et al. Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn’s disease. Nature 411, 599–603 (2001).

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