NF-κB-HOTAIR axis links DNA damage response, chemoresistance and cellular senescence in ovarian cancer-Reference-Cited by-同舟云学术

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NF-κB-HOTAIR axis links DNA damage response, chemoresistance and cellular senescence in ovarian cancer

Published:2016-04-04 Issue:41 Volume:35 Page:5350-5361
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Özeş A R,Miller D F,Özeş O N,Fang F,Liu Y,Matei D,Huang T,Nephew K P

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc201675.pdf

Reference57 articles.

1. Zhang X-P, Liu F, Wang W . Regulation of the DNA damage response by p53 cofactors. Biophys J 2012; 102: 2251–2260.

2. Chien Y, Scuoppo C, Wang X, Fang X, Balgley B, Bolden JE et al. Control of the senescence-associated secretory phenotype by NF-κB promotes senescence and enhances chemosensitivity. Genes Dev 2011; 25: 2125–2136.

3. Wang J, Jacob NK, Ladner KJ, Beg A, Perko JD, Tanner SM et al. RelA/p65 functions to maintain cellular senescence by regulating genomic stability and DNA repair. EMBO Rep 2009; 10: 1272–1278.

4. Wu Z-H, Shi Y, Tibbetts RS, Miyamoto S . Molecular linkage between the kinase ATM and NF-κB signaling in response to genotoxic stimuli. Science 2006; 311: 1141–1146.

5. Janssens S, Tinel A, Lippens S, Tschopp J . PIDD mediates NF-κB activation in response to DNA damage. Cell 2005; 123: 1079–1092.

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