RelA/p65 functions to maintain cellular senescence by regulating genomic stability and DNA repair-Reference-Cited by-同舟云学术

RelA/p65 functions to maintain cellular senescence by regulating genomic stability and DNA repair

Published:2009-09-25 Issue:11 Volume:10 Page:1272-1278
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Rep

Author:

Wang Jingxin¹,Jacob Naduparambil K¹²,Ladner Katherine J¹,Beg Amer³,Perko James D¹,Tanner Stephan M¹,Liyanarachchi Sandya¹,Fishel Richard¹²⁴,Guttridge Denis C¹²⁴

Affiliation:

1. Human Cancer Genetics Program, and Immunology and Medical Genetics, The Ohio State University Columbus Ohio 43210 USA

2. Department of Molecular Virology, Immunology and Medical Genetics, The Ohio State University Columbus Ohio 43210 USA

3. Department of Interdisciplinary Oncology, H. Lee Moffitt Cancer Center and Research Institute at USF Tampa Florida 33612 USA

4. Arthur G. James Comprehensive Cancer Center, The Ohio State University Columbus Ohio 43210 USA

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1038/embor.2009.197

Reference29 articles.

1. Reversal of human cellular senescence: roles of the p53 and p16 pathways

2. Involvement of Rel/Nuclear Factor-κB Transcription Factors in Keratinocyte Senescence

3. Oncogene-induced senescence as an initial barrier in lymphoma development

4. Cellular senescence: when bad things happen to good cells

5. Crucial role of p53-dependent cellular senescence in suppression of Pten-deficient tumorigenesis

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