Ataxin-1 with an expanded glutamine tract alters nuclear matrix-associated structures-Reference-Cited by-同舟云学术

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Ataxin-1 with an expanded glutamine tract alters nuclear matrix-associated structures

Published:1997-10-30 Issue:6654 Volume:389 Page:971-974
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Skinner Pamela J.,Koshy Beena T.,Cummings Christopher J.,Klement Ivan A.,Helin Kara,Servadio Antonio,Zoghbi Huda Y.,Orr Harry T.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/40153.pdf

Reference29 articles.

1. 1. Zoghbi, H. Y. & Orr, H. T. Spinocerebellar ataxia type 1. Semin. Cell Biol. 6, 29–35 (1995).

2. 2. Ross, C. A. When more is less: Pathogenesis of glutamine repeat neurodegenerative diseases. Neuron 15, 493–496 (1995).

3. 3. Burright, E. N. et al. SCA1 transgenic mice: a model for neurodegeneration caused by an expanded CAG trinucleotide repeat. Cell 82, 937–948 (1995).

4. 4. Stuurman, N. et al. Amonoclonal antibody recognizing nuclear matrix-associated nuclear bodies. J. Cell Sci. 101, 773–784 (1992).

5. 5. Weis, K. et al. Retinoic acid regulates aberrant nuclear localization of PML-RAR alpha in acute promyelocytic leukemia cells. Cell 76, 345–356 (1994).

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