NOTCH1 mediates a switch between two distinct secretomes during senescence-Reference-Cited by-同舟云学术

NOTCH1 mediates a switch between two distinct secretomes during senescence

Published:2016-08-15 Issue:9 Volume:18 Page:979-992
ISSN:1465-7392
Container-title:Nature Cell Biology
language:en
Short-container-title:Nat Cell Biol

Author:

Hoare Matthew,Ito Yoko,Kang Tae-Won,Weekes Michael P.,Matheson Nicholas J.,Patten Daniel A.,Shetty Shishir,Parry Aled J.,Menon Suraj,Salama Rafik,Antrobus Robin,Tomimatsu Kosuke,Howat William,Lehner Paul J.,Zender Lars,Narita Masashi

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology

Link

http://www.nature.com/articles/ncb3397.pdf

Reference66 articles.

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3. Childs, B. G., Durik, M., Baker, D. J. & van Deursen, J. M. Cellular senescence in aging and age-related disease: from mechanisms to therapy. Nat. Med. 21, 1424–1435 (2015).

4. Acosta, J. C. et al. A complex secretory program orchestrated by the inflammasome controls paracrine senescence. Nat. Cell Biol. 15, 978–990 (2013).

5. Hubackova, S., Krejcikova, K., Bartek, J. & Hodny, Z. IL1- and TGFβ-Nox4 signaling, oxidative stress and DNA damage response are shared features of replicative, oncogene-induced, and drug-induced paracrine ‘Bystander senescence’. Aging (Albany NY) 4, 932–951 (2012).

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