A complex secretory program orchestrated by the inflammasome controls paracrine senescence-Reference-Cited by-同舟云学术

A complex secretory program orchestrated by the inflammasome controls paracrine senescence

Published:2013-06-16 Issue:8 Volume:15 Page:978-990
ISSN:1465-7392
Container-title:Nature Cell Biology
language:en
Short-container-title:Nat Cell Biol

Author:

Acosta Juan Carlos,Banito Ana,Wuestefeld Torsten,Georgilis Athena,Janich Peggy,Morton Jennifer P.,Athineos Dimitris,Kang Tae-Won,Lasitschka Felix,Andrulis Mindaugas,Pascual Gloria,Morris Kelly J.,Khan Sadaf,Jin Hong,Dharmalingam Gopuraja,Snijders Ambrosius P.,Carroll Thomas,Capper David,Pritchard Catrin,Inman Gareth J.,Longerich Thomas,Sansom Owen J.,Benitah Salvador Aznar,Zender Lars,Gil Jesús

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology

Link

http://www.nature.com/articles/ncb2784.pdf

Reference53 articles.

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2. Campisi, J. & d’Adda di Fagagna, F. Cellular senescence: when bad things happen to good cells. Nat. Rev. Mol. Cell Biol. 8, 729–740 (2007).

3. Kuilman, T. & Peeper, D.S. Senescence-messaging secretome: SMS-ing cellular stress. Nat. Rev. Cancer 9, 81–94 (2009).

4. Coppe, J. P. et al. A human-like senescence-associated secretory phenotype is conserved in mouse cells dependent on physiological oxygen. PLoS One 5, e9188 (2010).

5. Krtolica, A., Parrinello, S., Lockett, S., Desprez, P. Y. & Campisi, J. Senescent fibroblasts promote epithelial cell growth and tumorigenesis: a link between cancer and aging. Proc. Natl Acad. Sci. USA 98, 12072–12077 (2001).

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