Lack of CD45 in FLT3-ITD mice results in a myeloproliferative phenotype, cortical porosity, and ectopic bone formation-Reference-Cited by-同舟云学术

Lack of CD45 in FLT3-ITD mice results in a myeloproliferative phenotype, cortical porosity, and ectopic bone formation

Published:2019-02-28 Issue:24 Volume:38 Page:4773-4787
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Kresinsky Anne,Schnöder Tina M.,Jacobsen Ilse D.,Rauner Martina,Hofbauer Lorenz C.,Ast Volker,König Rainer,Hoffmann Bianca,Svensson Carl-Magnus,Figge Marc Thilo,Hilger Ingrid,Heidel Florian H.,Böhmer Frank- D.,Müller Jörg P.

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/s41388-019-0757-y.pdf

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3. Pinheiro RF, Moreira Ede S, Silva MR, Greggio B, Alberto FL, Chauffaille Mde L. FLT3 mutation and AML/ETO in a case of Myelodysplastic syndrome in transformation corroborates the two hit model of leukemogenesis. Leukemia Res. 2007;31:1015–8.

4. Stirewalt DL, Radich JP. The role of FLT3 in haematopoietic malignancies. Nat Rev Cancer. 2003;3:650–65.

5. Mizuki M, Fenski R, Halfter H, Matsumura I, Schmidt R, Müller C. et al. Flt3 mutations from patients with acute myeloid leukemia induce transformation of 32D cells mediated by the Ras and STAT5 pathways. Blood. 2000;96:3907–14.

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