LEDGF (p75) promotes DNA-end resection and homologous recombination-Reference-Cited by-同舟云学术

LEDGF (p75) promotes DNA-end resection and homologous recombination

Published:2012-07-08 Issue:8 Volume:19 Page:803-810
ISSN:1545-9993
Container-title:Nature Structural & Molecular Biology
language:en
Short-container-title:Nat Struct Mol Biol

Author:

Daugaard Mads,Baude Annika,Fugger Kasper,Povlsen Lou Klitgaard,Beck Halfdan,Sørensen Claus Storgaard,Petersen Nikolaj H T,Sorensen Poul H B,Lukas Claudia,Bartek Jiri,Lukas Jiri,Rohde Mikkel,Jäättelä Marja

Publisher

Springer Science and Business Media LLC

Subject

Molecular Biology,Structural Biology

Link

http://www.nature.com/articles/nsmb.2314.pdf

Reference59 articles.

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3. Ge, H., Si, Y. & Wolffe, A.P. A novel transcriptional coactivator, p52, functionally interacts with the essential splicing factor ASF/SF2. Mol. Cell 2, 751–759 (1998).

4. Brown-Bryan, T.A. et al. Alternative splicing and caspase-mediated cleavage generate antagonistic variants of the stress oncoprotein LEDGF/p75. Mol. Cancer Res. 6, 1293–1307 (2008).

5. Nishizawa, Y., Usukura, J., Singh, D.P., Chylack, L.T. Jr. & Shinohara, T. Spatial and temporal dynamics of two alternatively spliced regulatory factors, lens epithelium-derived growth factor (ledgf/p75) and p52, in the nucleus. Cell Tissue Res. 305, 107–114 (2001).

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