Mutations in a member of the ADAMTS gene family cause thrombotic thrombocytopenic purpura-Reference-Cited by-同舟云学术

Mutations in a member of the ADAMTS gene family cause thrombotic thrombocytopenic purpura

Published:2001-10-04 Issue:6855 Volume:413 Page:488-494
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Levy Gallia G.,Nichols William C.,Lian Eric C.,Foroud Tatiana,McClintick Jeanette N.,McGee Beth M.,Yang Angela Y.,Siemieniak David R.,Stark Kenneth R.,Gruppo Ralph,Sarode Ravindra,Shurin Susan B.,Chandrasekaran Visalam,Stabler Sally P.,Sabio Hernan,Bouhassira Eric E.,Upshaw Jefferson D.,Ginsburg David,Tsai Han-Mou

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

https://www.nature.com/articles/35097008.pdf

Reference40 articles.

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2. George, J. N. How I treat patients with thrombotic thrombocytopenic purpura-hemolytic uremic syndrome. Blood 96, 1223–1229 (2000).

3. Rock, G. A. et al. Comparison of plasma exchange with plasma infusion in the treatment of thrombotic thrombocytopenic purpura. N. Engl. J. Med. 325, 393–397 (1991).

4. Moake, J. L. et al. Unusually large plasma factor VIII: von Willebrand factor multimers in chronic relapsing thrombotic thrombocytopenic purpura. N. Engl. J. Med. 307, 1432–1435 (1982).

5. Tsai, H. M. Physiologic cleavage of von Willebrand factor by a plasma protease is dependent on its conformation and requires calcium ion. Blood 87, 4235–4244 (1996).

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