Desialylation is a mechanism of Fc-independent platelet clearance and a therapeutic target in immune thrombocytopenia-Reference-Cited by-同舟云学术

Desialylation is a mechanism of Fc-independent platelet clearance and a therapeutic target in immune thrombocytopenia

Published:2015-07-17 Issue:1 Volume:6 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Li June,van der Wal Dianne E.,Zhu Guangheng,Xu Miao,Yougbare Issaka,Ma Li,Vadasz Brian,Carrim Naadiya,Grozovsky Renata,Ruan Min,Zhu Lingyan,Zeng Qingshu,Tao Lili,Zhai Zhi-min,Peng Jun,Hou Ming,Leytin Valery,Freedman John,Hoffmeister Karin M.,Ni Heyu

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms8737.pdf

Reference70 articles.

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3. Cines, D. B., Bussel, J. B., Liebman, H. A. & Luning Prak, E. T. The ITP syndrome: pathogenic and clinical diversity. Blood 113, 6511–6521 (2009).

4. Mathias, S. D. et al. Impact of chronic Immune Thrombocytopenic Purpura (ITP) on health-related quality of life: a conceptual model starting with the patient perspective. Health Qual. Life Outcomes 6, 13 (2008).

5. Provan, D. et al. International consensus report on the investigation and management of primary immune thrombocytopenia. Blood 115, 168–186 (2010).

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