The kinase LRRK2 is a regulator of the transcription factor NFAT that modulates the severity of inflammatory bowel disease-Reference-Cited by-同舟云学术

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The kinase LRRK2 is a regulator of the transcription factor NFAT that modulates the severity of inflammatory bowel disease

Published:2011-10-09 Issue:11 Volume:12 Page:1063-1070
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Liu Zhihua,Lee Jinwoo,Krummey Scott,Lu Wei,Cai Huaibin,Lenardo Michael J

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/ni.2113.pdf

Reference48 articles.

1. Abraham, C. & Cho, J.H. Inflammatory bowel disease. N. Engl. J. Med. 361, 2066–2078 (2009).

2. Barrett, J.C. et al. Genome-wide association defines more than 30 distinct susceptibility loci for Crohn′s disease. Nat. Genet. 40, 955–962 (2008).

3. Franke, A. et al. Genome-wide meta-analysis increases to 71 the number of confirmed Crohn′s disease susceptibility loci. Nat. Genet. 42, 1118–1125 (2010).

4. Anderson, C.A. et al. Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47. Nat. Genet. 43, 246–252 (2011).

5. Kaser, A. & Blumberg, R.S. Endoplasmic reticulum stress in the intestinal epithelium and inflammatory bowel disease. Semin. Immunol. 21, 156–163 (2009).

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