KIT Is a Frequent Target for Epigenetic Silencing in Cutaneous Melanoma-Reference-Cited by-同舟云学术

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KIT Is a Frequent Target for Epigenetic Silencing in Cutaneous Melanoma

Published:2015-02 Issue:2 Volume:135 Page:516-524
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Dahl Christina,Abildgaard Cecilie,Riber-Hansen Rikke,Steiniche Torben,Lade-Keller Johanne,Guldberg Per

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference47 articles.

1. Somatic activation of KIT in distinct subtypes of melanoma;Curtin;J Clin Oncol,2006

2. Mutual exclusivity analysis of genetic and epigenetic drivers in melanoma identifies a link between p14ARF and RARbeta signaling;Dahl;Mol Cancer Res,2013

3. The genome and epigenome of malignant melanoma;Dahl;APMIS,2007

4. Comparison of Beta-value and M-value methods for quantifying methylation levels by microarray analysis;Du;BMC Bioinformatics,2010

5. Gene Expression Omnibus: NCBI gene expression and hybridization array data repository;Edgar;Nucleic Acids Res,2002

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