Bcl-xL promotes metastasis independent of its anti-apoptotic activity-Reference-Cited by-同舟云学术

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Bcl-xL promotes metastasis independent of its anti-apoptotic activity

Published:2016-01-20 Issue:1 Volume:7 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Choi Soyoung,Chen Zhengming,Tang Laura H.,Fang Yuanzhang,Shin Sandra J.,Panarelli Nicole C.,Chen Yao-Tseng,Li Yi,Jiang Xuejun,Du Yi-Chieh Nancy

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms10384.pdf

Reference57 articles.

1. Hanahan, D. & Weinberg, R. A. Hallmarks of cancer: the next generation. Cell 144, 646–674 (2011).

2. Reed, J. C. Dysregulation of apoptosis in cancer. J. Clin. Oncol. 17, 2941–2953 (1999).

3. Yin, X. M., Oltvai, Z. N. & Korsmeyer, S. J. BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax. Nature 369, 321–323 (1994).

4. Placzek, W. J. et al. A survey of the anti-apoptotic Bcl-2 subfamily expression in cancer types provides a platform to predict the efficacy of Bcl-2 antagonists in cancer therapy. Cell Death Dis. 1, e40 (2010).

5. Davids, M. S. & Letai, A. Targeting the B-cell lymphoma/leukemia 2 family in cancer. J. Clin. Oncol. 30, 3127–3135 (2012).

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