ONC212, alone or in synergistic conjunction with Navitoclax (ABT-263), promotes cancer cell apoptosis via unconventional mitochondrial-independent caspase-3 activation-Reference-Cited by-同舟云学术

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ONC212, alone or in synergistic conjunction with Navitoclax (ABT-263), promotes cancer cell apoptosis via unconventional mitochondrial-independent caspase-3 activation

Published:2024-09-13 Issue:1 Volume:22 Page:
ISSN:1478-811X
Container-title:Cell Communication and Signaling
language:en
Short-container-title:Cell Commun Signal

Author:

Basu Vishal,Shabnam ,Murghai Yamini,Ali Maqsood,Sahu Swetangini,Verma Bhupendra K.,Seervi Mahendra

Funder

DST-SERB

All-India Institute of Medical Sciences

Publisher

Springer Science and Business Media LLC

Link

https://link.springer.com/content/pdf/10.1186/s12964-024-01817-1.pdf

Reference69 articles.

1. Warburg O. On the Origin of Cancer Cells. Science. 1956;123:309–14.

2. Hanahan D. Hallmarks of Cancer: New Dimensions. Cancer Discov. 2022;12:31–46.

3. Green DR, Reed JC. Mitochondria and Apoptosis. Science. 1998;281:1309–12.

4. Dong L, Gopalan V, Holland O, Neuzil J. Mitocans Revisited: Mitochondrial Targeting as Efficient Anti-Cancer Therapy. Int J Mol Sci. 2020;21:7941.

5. Boukalova S, Rohlenova K, Rohlena J, Neuzil J. Mitocans: Mitochondrially Targeted Anti-cancer Drugs. Mitochondrial Biology and Experimental Therapeutics. 2018. p. 613–35.

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