Uncompromised NK cell activation is essential for virus-specific CTL activity during acute influenza virus infection-Reference-Cited by-同舟云学术

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Uncompromised NK cell activation is essential for virus-specific CTL activity during acute influenza virus infection

Published:2017-04-17 Issue:9 Volume:15 Page:827-837
ISSN:1672-7681
Container-title:Cellular & Molecular Immunology
language:en
Short-container-title:Cell Mol Immunol

Author:

Liu Yuan,Zheng Jian,Liu Yinping,Wen Liyan,Huang Lei,Xiang Zheng,Lam Kwok-Tai,Lv Aizhen,Mao Huawei,Lau Yu-Lung,Tu Wenwei

Publisher

Springer Science and Business Media LLC

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/cmi201710.pdf

Reference43 articles.

1. Medina RA, Garcia-Sastre A. Influenza A viruses: new research developments. Nat Rev Microbiol 2011; 9: 590–603.

2. Duggal NK, Emerman M. Evolutionary conflicts between viruses and restriction factors shape immunity. Nat Rev Immunol 2012; 12: 687–695.

3. Schmolke M, Garcia-Sastre A. Evasion of innate and adaptive immune responses by influenza A virus. Cell Microbiol 2010; 12: 873–880.

4. Varga ZT, Grant A, Manicassamy B, Palese P. Influenza virus protein PB1-F2 inhibits the induction of type I interferon by binding to MAVS and decreasing mitochondrial membrane potential. J Virol 2012; 86: 8359–8366.

5. Conenello GM, Tisoncik JR, Rosenzweig E, Varga ZT, Palese P, Katze MG. A single N66S mutation in the PB1-F2 protein of influenza A virus increases virulence by inhibiting the early interferon response in vivo. J Virol 2011; 85: 652–662.

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