Perinatal angiogenesis from pre-existing coronary vessels via DLL4–NOTCH1 signalling
Author:
Funder
American Heart Association
U.S. Department of Health & Human Services | National Institutes of Health
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology
Link
https://www.nature.com/articles/s41556-021-00747-1.pdf
Reference44 articles.
1. Del Monte, G. & Harvey, R. P. An endothelial contribution to coronary vessels. Cell 151, 932–934 (2012).
2. Wu, B. et al. Endocardial cells form the coronary arteries by angiogenesis through myocardial-endocardial VEGF signaling. Cell 151, 1083–1096 (2012).
3. Wang, Y. et al. Uncontrolled angiogenic precursor expansion causes coronary artery anomalies in mice lacking Pofut1. Nat. Commun. 8, 578 (2017).
4. Zhang, Z. & Zhou, B. Accelerated coronary angiogenesis by Vegfr1-knockout endocardial cells. PLoS ONE 8, e70570 (2013).
5. Red-Horse, K., Ueno, H., Weissman, I. L. & Krasnow, M. A. Coronary arteries form by developmental reprogramming of venous cells. Nature 464, 549–553 (2010).
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