PI3Kα-regulated gelsolin activity is a critical determinant of cardiac cytoskeletal remodeling and heart disease-Reference-Cited by-同舟云学术

PI3Kα-regulated gelsolin activity is a critical determinant of cardiac cytoskeletal remodeling and heart disease

Published:2018-12 Issue:1 Volume:9 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Patel Vaibhav B.^ORCID,Zhabyeyev Pavel^ORCID,Chen Xueyi,Wang Faqi,Paul Manish,Fan Dong,McLean Brent A.,Basu Ratnadeep,Zhang Pu,Shah Saumya,Dawson John F.,Pyle W. Glen,Hazra Mousumi^ORCID,Kassiri Zamaneh,Hazra Saugata,Vanhaesebroeck Bart,McCulloch Christopher A.^ORCID,Oudit Gavin Y.

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/s41467-018-07812-8.pdf

Reference72 articles.

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3. Yancy, C. W. et al. 2013 ACCF/AHA guideline for the management of heart failure: executive summary: a report of the American College of Cardiology Foundation/American Heart Association Task Force on practice guidelines. Circulation 128, 1810–1852 (2013).

4. Guo, D. et al. Loss of PI3Kgamma enhances cAMP-dependent MMP remodeling of the myocardial N-cadherin adhesion complexes and extracellular matrix in response to early biomechanical stress. Circ. Res. 107, 1275–1289 (2010).

5. Patel, V. B. et al. Loss of p47phox subunit enhances susceptibility to biomechanical stress and heart failure because of dysregulation of cortactin and actin filaments. Circ. Res. 112, 1542–1556 (2013).

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