Loss of p47 phox Subunit Enhances Susceptibility to Biomechanical Stress and Heart Failure Because of Dysregulation of Cortactin and Actin Filaments-Reference-Cited by-同舟云学术

Loss of p47 phox Subunit Enhances Susceptibility to Biomechanical Stress and Heart Failure Because of Dysregulation of Cortactin and Actin Filaments

Published:2013-06-07 Issue:12 Volume:112 Page:1542-1556
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circ Res

Author:

Patel Vaibhav B.¹,Wang Zuocheng¹,Fan Dong¹,Zhabyeyev Pavel¹,Basu Ratnadeep¹,Das Subhash K.¹,Wang Wang¹,DesAulniers Jessica¹,Holland Steven M.¹,Kassiri Zamaneh¹,Oudit Gavin Y.¹

Affiliation:

1. From the Department of Medicine, Division of Cardiology (V.B.P., Z.W., P.Z., S.K.D., J.D.A., G.Y.O.), Mazankowski Alberta Heart Institute (V.B.P., Z.W., D.F., P.Z., R.B., S.K.D., W.W., J.D.A., Z.K., G.Y.O.), and Department of Physiology (D.F., R.B., W.W., Z.K., G.Y.O.), University of Alberta, Edmonton, AB, Canada; and National Institute of Allergy and Infectious Diseases, Bethesda, MD (S.M.H.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference64 articles.

1. Biochemistry, Physiology, and Pathophysiology of NADPH Oxidases in the Cardiovascular System

2. Phosphorylation of p47phox directs phox homology domain from SH3 domain toward phosphoinositides, leading to phagocyte NADPH oxidase activation

3. A specific p47phox -serine phosphorylated by convergent MAPKs mediates neutrophil NADPH oxidase priming at inflammatory sites

4. Phosphorylation of p47phox Sites by PKC α, βΙΙ, δ, and ζ: Effect on Binding to p22phox and on NADPH Oxidase Activation

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